Although both type I and type II hypersensitivities involve antibodies as immune effectors, different mechanisms are involved with these different hypersensitivities. Differentiate the two.
What types of antibodies are most common in type III hypersensitivities, and why?
Why is a parent usually a better match for transplanted tissue to a donor than a random individual of the same species?
Compare the treatments for primary and secondary immunodeficiencies.
How can tumor antigens be effectively targeted without inducing an autoimmune (anti-self) response?